CAS 11097-68-0PubChem CID 23053647
Aluminum Sesquichlorohydrate
- IUPAC name
- aluminum;chloride;hydroxide;hydrate
- Molecular formula
- AlClH3O2+
- Molecular weight
- 97.46 g/mol
- Exact mass
- 96.9636954 Da
- Topological polar surface area
- 2.0 Ų
- Hydrogen-bond donors
- 2
- Hydrogen-bond acceptors
- 3
- Covalent units
- 4
- Defined stereocentres
- 0
Also known as
ALUMINUM CHLORIDE HYDROXIDEHydrofugalAloxicollAstringenGelsicaSansudorBanoltan WhiteHessidrex WT
16 more reported names
Cartafix LAAluminol ACHBerukotan AC-PAquarhone 18Astringen 10Aluminum chlorohydrolKemwater PAX 14Kempac 10Kempac 20Wickenol cps 325OCALOulupac 180Paho 2SPALCPAC (salt)Chlorhydrol Micro-Dry
Evidence from additional scientific databases
NIH PubChem PUG-View
Exact identifier match23053647
Attributed physical-property, hazard, environmental and use annotations.
- Carcinogen Classification: Not listed by IARC. IARC classified aluminum production as carcinogenic to humans (Group 1), but did not implicate aluminum itself as a human carcinogen. (L135) A link between use of aluminum-containing antiperspirants and increased risk of breast cancer has been proposed (A235), but studies have not been able to establish a clear link (A15468). Source: Toxin and Toxin Target Database (T3DB)
- Exposure Routes: Oral (L739) ; inhalation (L739) Source: Toxin and Toxin Target Database (T3DB)
- Health Effects: Aluminum targets the nervous system and causes decreased nervous system performance and is associated with altered function of the blood-brain barrier. The accumulation of aluminum in the body may cause bone or brain diseases. High levels of aluminum have been linked to Alzheimer's disease. A small percentage of people are allergic to aluminium and experience contact dermatitis, digestive disorders, vomiting or other symptoms upon contact or ingestion of products containing aluminium. (L739, L740) Source: Toxin and Toxin Target Database (T3DB)
- Toxicity Summary: The main target organs of aluminum are the central nervous system and bone. Aluminum binds with dietary phosphorus and impairs gastrointestinal absorption of phosphorus. The decreased phosphate body burden results in osteomalacia (softening of the bones due to defective bone mineralization) and rickets. Aluminum's neurotoxicity is believed to involve several mechanisms. Changes in cytoskeletal protein functions as a results of altered phosphorylation, proteolysis, transport, and synthesis are believed to be one cause. Aluminum may induce neurobehavioral effects by affecting permeability of the blood-brain barrier, cholinergic activity, signal transduction pathways, lipid peroxidation, and impair neuronal glutamate nitric oxide-cyclic GMP pathway, as well as interfere with metabolism of essential trace elements because of similar coordination chemistries and consequent competitive interactions. It has been suggested that aluminum's interaction with estrogen receptors increases the expression of estrogen-related genes and thereby contributes to the progression of breast cancer (A235), but studies have not been able to establish a clear link between aluminum and increased risk of brea Source: Toxin and Toxin Target Database (T3DB)
- Uses: Aluminium chlorohydrate is used in deodorants and antiperspirants and as a flocculant in water purification. (L746) Source: Toxin and Toxin Target Database (T3DB)
Mapped by: Existing checksum-valid CAS to unique PubChem CID match. Retrieved: 2026-08-31. Open source record
Scientific classifications and hazard annotations provide context and do not replace the market-specific regulatory decision shown above.
Additional source coverage and match status
| Source | Result | Checked |
|---|---|---|
| NIH PubChem PUG-View | Exact match | 2026-08-31 |
A recorded no-match is useful evidence that the source was checked; it is not a claim that the substance does not exist elsewhere.
Retrieved from NIH PubChem on 2026-08-31 by checksum-valid CAS matching. Chemical properties do not replace the regulatory decision on this page.